| Lumbar
spinal stenosis is a result of aging and everyday wear and
tear on the spine. Though these changes happen to all of us, not
everyone will experience the symptoms of lumbar
spinal stenosis (LSS). Features of LSS include persistent,
progressive low back pain with or without radiation, numbness or
weakness in the buttocks and legs, and symptoms that improve
with resting, lying down or bending forward. Unfortunately, many
patients with symptoms of
spinal stenosis often suffer for a long period of time
before seeking medical advice and, even then, can often be
misdiagnosed and treated inappropriately. As such, many older
people with LSS have had to give up active lifestyles despite
conservative treatment with rest, medication, and physical
therapy.
What Happens in Lumbar Stenosis?
In the normal spinal canal, the nerve roots are contained in
the thecal sac and exit between each vertebra (foramina). The
central spinal canal and the foramen contain the thecal sac and
nerve roots respectively.
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Figure 1
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The canal and foramen are formed (Figure 1) by bony
structures (vertebral body, facets, pedicles) as well as
soft tissue structures (ligamentum flavum, facet capsules,
intervertebral disc annulus). As we age, degenerative
changes occur in our spine causing narrowing of the central
spinal canal area, and/or the area where the nerve roots
exit the spinal canal (foramina). One of these degenerative
changes is the formation of extra bony growths called
osteophytes. Additionally, the large connective "band" (ligamentum
flavum) which runs along the inside the spinal canal can
become coarse and thickened also causing narrowing of the
central canal space. |
How do you get Lumbar Stenosis?
Though we don't know why some people develop symptoms of
spinal stenosis and others do not, we do know that some
people are born with the predisposition towards it. In normal
development, the spinal canal reaches adult size by about age
four. If the canal does not reach adequate size by this age,
then it will never "catch up" and
spinal stenosis results and occurs uniformly throughout the
spine (Figure 2a).
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Figure 2a
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With this type of stenosis, you may remain symptom free until
other conditions further compromise the canal space. Such
conditions include formation of extra bony growths (osteophytes),
trauma, or intervertebral disc problems. Others may develop
stenosis without this predisposition but can instead "acquire"
it through trauma, degenerative changes (osteophytes), bulging
of the large connective "band" (ligamentum flavum), and most
commonly by spondylosis.
As we age our body begins to dehydrate. This process causes
our intervertebral discs to dry out thus decreasing the area of
cushioning between each vertebra. Normally our discs separate
each vertebra, act as shock absorbers during regular activity,
and allow our spine to move freely. A decrease in the area of
cushioning leads to trauma of our vertebrae and formation of
osteophytes. This condition is referred to as Spondylosis.
Several of these changes are demonstrated in Figure 2b.
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Figure 2b
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Figure 2c demonstrates changes from the normal anatomy to
those found in lumbar and foraminal; stenosis.
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Figure 2c
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The vertebrae are connected in the back by the bony facets
and intervening pars interarticularis. These structures in
combination with the intervertebral disc help to interlock each
level of the spine. Degeneration, spondylosis, and some
congenital conditions can cause these joints to progressively
fail. Eventually, a vertebra can slip forward and compromise
either the central canal space and/or the opening through which
the nerve root exits the spinal canal (foramina). This condition
is called
spondylolisthesis (Figure 3). It is more prominent in groups
of people with who place a lot of stress on their backs
including manual laborers, heavy machine operators, and
professional athletes.
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Figure 3
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Symptoms: What to Look For
LSS can cause compression of the spinal nerves in the lumbar
area (cauda equina). At the same time, the cauda equina between
the levels of stenosis becomes congested due to alterations in
the blood vessels that drain blood from the area. This
congestion prevents the nerve roots from conducting effectively,
especially when walking. When this happens, there can be a
significant amount of pain, numbness, and/or weakness in the
buttocks, thighs, and legs. Often, this can severely limit or
altogether stop the patient from walking. The discomfort usually
disappears after 5 -10 minutes of rest. Lying with the legs
flexed, sitting, or squatting can also help as these maneuvers
increase the area of the spinal canal. These symptoms may begin
in the lower legs and progress upwards toward the buttocks or
they may begin in the buttocks and progress downward. This is
referred to a "sensory march." Low back pain is also a very
common complaint. The symptoms may begin on one side but will
often end up involving both sides. You may hear your doctor
refer to this as neurogenic claudication.
Neurogenic claudication tends to occur more frequently in men
than women and is usually seen after age 50. Most people will
gradually decrease the walking distance until they reach a
comfort zone. Typically, patients are able to walk at least 100
meters. Unfortunately, this condition is lifelong and can be
progressive. Sometimes physical therapy, medical pain management
and other non-surgical measures can provide adequate symptomatic
relief. If your symptoms continue to progress or become too
painful, surgery to widen the spinal canal may be your best
option (surgical decompression).
It is important for your physician to differentiate
neurogenic claudication from decreased blood flow to the lower
extremities due to calcified blood vessels (peripheral vascular
disease) since this condition also often occurs in older people
and has similar symptoms. However, symptoms of peripheral
vascular disease typically are not relieved by changes in
posture and do not exhibit any of the "sensory march" symptoms.
LSS can also cause pain extending down the leg along the area
that corresponds to the affected nerve root. This occurs because
the area where the nerve root exits the spinal canal (foramina)
has become narrowed causing pressure on the nerve root.
Foraminal stenosis is thus a form of LSS and can coexist with
central stenosis and classic claudication symptoms. Often,
foraminal stenosis does respond well to conservative treatment
without surgery, but may require 6 to12 months for recovery.
However, those with persistent severe pain will likely benefit
from surgery to widen the foraminal space (surgical
decompression).
Diagnostic Studies: X-rays, CT Scan, MRI, Myelography
Diagnostic studies are usually performed if symptoms do not
subside after a period of 3 to 6 months of therapy such as rest,
anti-inflammatory medications, and physical therapy. X-rays are
ordered cautiously because many people who do not have any
symptoms of spinal stenosis have abnormal x-rays, CT scans, and
MRI's. Surgery should only be performed in patients whose
symptoms correlate with findings on these studies and a history
that supports these findings.
Typically, plain x-rays are done first. They are helpful in
looking for infection, tumors, and identifying problems with
alignment of the spine (Figure 4a).
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Figure 4a
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But X-rays will not always identify LSS. CT scans have become
the most widely used test for evaluating the spine because it is
noninvasive and provides three-dimensional views of the spine.
It is also particularly helpful in differentiating between hard
tissue (bony osteophytes) and soft tissue (intervertebral
discs). Myelography involves injection of dye into the spinal
sac where is mixes with the cerebrospinal fluid. Any outward
protrusion will block the flow of the dye. CT scans done in
conjunction with myelography are particularly helpful diagnosing
LSS (Figure 4b).
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Figure 4b
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The MRI is the newest non-invasive diagnostic test for
evaluating the spine and offers more precise description of disc
disease, soft tissue changes and the degree of stenosis (Figure
4c).
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Figure 4c
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Treatment Options: Medication, Physical Therapy, Surgery
Once the diagnosis of LSS is made, you and your doctor will
decide on the most appropriate therapy including medications,
physical therapy, and surgery. Many patients will respond to
non-surgical interventions. Anti-inflammatory medications such
as ibuprofen and acetaminophen can help reduce symptoms in the
earlier stages of LSS. Cortisone injections at the site of
low back pain can also help reduce inflammation of the
spinal nerves and nerve roots to help control pain. Usually no
more than three cortisone injections per year are recommended.
However, these are temporary relief measures and symptoms may
resurface once the medication wears off.
In addition to medications, physical therapy can help to
restore flexibility and strengthen back and abdominal muscles to
provide substantial relief from symptoms of LSS. If non-surgical
measures do not offer adequate relief, your doctor may recommend
back surgery.
Who is a Candidate for Surgery to Relieve LSS?
Patients who are generally in good health should have no
problem undergoing surgery. Age alone is not a major limiting
factor however, if you have other medical conditions such as
high blood pressure, or diabetes that typically accompany old
age, surgery may pose greater risk.
Surgical Procedures
The operations typically used to treat Lumbar Stenosis
include the classic laminectomy, laminotomy, and foraminotomy.
For patients who meet the proper indications, these procedures
can also be combined with a spinal fusion operation. The most
commonly used decompressive surgery is the laminectomy. To
perform a classic laminectomy a 3-4 inch incision is made in
your lower back (Figure 5a), though it may be longer depending
on how many levels of your lamina are affected.
A laminectomy involves removing the bony extensions (lamina)
from the backside of the vertebral body which are causing
pressure on the spinal sac and/or the nerve roots (Figure 5c-e).
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Figure 5c
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Figure 5d
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Figure 5e
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Often, only a portion of the lamina needs to be removed to
relieve the pressure on the nerve roots (laminotomy). The
ligaments (ligamentum flavum) and soft-tissue (facet capsules,
herniated or bulging discs) in the affected area are also
removed to increase the canal space. At the same time, a portion
of the facet joints at the sides of the lamina may also be
removed since they also cause increased pressure on the central
and foraminal areas (Figure 5b).
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Figure 5b
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The goal of a foraminotomy is to enlarge the space where the
nerve roots exit the spinal canal thus decreasing pressure on
them. Foraminotomies can be done by themselves or often in
conjunction with a laminectomy.
Some patients may develop instability of the spine with
surgery. This occurs when a lot of bone needs to be removed
and/or when multiple levels are operated on in order to provide
adequate decompression. As such, many surgeons prefer more
limited lamina removal (laminotomy) and only partial facet
removal (medial facetectomy). Others may already have
instability from their disease as in cases of
spondylolisthesis. For all these patients, a spinal fusion
is needed in addition to decompression. Spinal fusion involves
grafting bone onto the spine and using instrumentation, such as
screws and rods, to support the spine and provide stability.
Your neurosurgeon can usually determine whether you will need a
fusion prior to surgery so that you will be able to discuss this
possibility beforehand.
Microendoscopic Laminectomy
The Micro-Endoscopic Laminotomy (MEL) is an exciting new
treatment option for patients who are candidates for the
surgical treatment of lumbar stenosis. MEL accomplishes the same
goal as the classical laminectomy but it involves using a
minimally-invasive approach, a state-of-the-art surgical
endoscope for visualization , and microsurgical decompressive
techniques.
A thin needle is placed under fluoroscopic (x-ray) guidance
down to the involved level on one side of the midline spine
(Figure 6a).
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Figure 6a
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A small ½ to 1-inch incision is then made around this needle.
Using a set of tapered metal dilators passed over the guiding
needle, the tissue and muscles are then gently spread off the
underlying bone (Figure 6b,c).
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Figure 6b
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Figure 6c
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A hollow metal cylinder is then passed down to the area of
the stenosis and secured. Through this working channel, a rigid
surgical micro-endoscopic camera is placed to provide your
surgeons with a close-up, magnified view of the pathology
(Figure 6d,e).
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Figure 6d
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With this close-up operative view, your surgeon
can then microsurgically remove the bone (Figure 6f) compressing
the nerve roots thereby relieving the stenosis.
In addition, soft tissue such as the ligamentum flavum
(Figure 6g) and
herniated discs can also be removed through the MEL
technique (Figure 6h). In our experience, excellent
decompression of both sides of the spinal canal can be achieved
through this one-sided approach.
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Figure 6g
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Figure 6h
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In the hands of experienced spinal, the same amount of
decompression can be achieved through the MEL technique as would
typically be obtained through open surgery (Figure 6i).
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Figure 6i
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Through the same incision, the surgeon can swing the
endoscope to decompress the level immediately above and below as
well. Overall when compared to traditional open procedures, the
MEL technique offers the attractive benefits of far less
disruption of normal tissue, faster surgical time, decreased
post-operative discomfort, quicker recovery time, and a
typically more rapid return to normal activity. You should
contact your surgeon to find out if the MEL operation is
available in your area and whether or not you are a candidate
for the procedure.
Your Recovery
You will be asleep during surgery under general anesthesia.
After surgery you will be taken to the recovery area where you
will be monitored until you awaken. Most patients can begin
getting out of bed on the same day surgery is performed.
Activity is gradually increased and patients are typically able
to go home within two to five days after a classical laminectomy
and one to two days after MEL. However, these time frames may be
longer depending on the extent your surgery.
Typically you will experience pain for a few weeks after
surgery and you may need over-the-counter or prescription pain
medications. Your neurosurgeon may also prescribe a course of
physical therapy to help you regain strength in your abdomen and
back and promote a better recovery. The total recovery time
after lumbar surgery can take anywhere from eight weeks to six
months, depending on the severity of your condition before
surgery as well as your overall health condition prior to
surgery. Common sense tells you the healthier you are the
quicker you will heal. As smoking weakens bones, accelerate
degeneration and slows the healing process, we also highly
recommend you stop before any type of surgical procedure.
Understanding the Risks
Decompressive laminectomy is the most common and successful
surgery done for treatment of symptoms associated with LSS. Over
60,000 such surgeries are done annually on patients over 60.
However, it is still surgery and any surgery involves risk. Your
neurosurgeon will discuss these risks in detail with you, but
the most common ones are bleeding, infection, injury to the
nerve, scarring, and the usual risks of anesthesia. Those with
accompanying chronic health conditions such as diabetes,
obesity, and high blood pressure or those with advanced stenosis
carry greater risk and may have poorer outcomes. Ultimately you
will be the one to decide which treatment option is best for
you. Your neurosurgeon will discuss all options with you and
explain the pros and cons of each to help you decide. Only you
can determine if your pain warrants a more definitive treatment
plan such as surgery. |